{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,10,7]],"date-time":"2026-10-07T08:51:18Z","timestamp":1791363078438,"version":"4.3.1"},"reference-count":15,"publisher":"Ovid Technologies (Wolters Kluwer Health)","issue":"4","content-domain":{"domain":["lww.com","ovid.com"],"crossmark-restriction":true},"short-container-title":[],"published-print":{"date-parts":[[2018,7]]},"abstract":"<jats:sec>\n                    <jats:title>Background:<\/jats:title>\n                    <jats:p>Pemphigus is an autoimmune blistering disease. The common cause of death in pemphigus is septicemia which is usually secondary to cutaneous bacterial infection.<\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>Aim:<\/jats:title>\n                    <jats:p>The aim was to study the cutaneous bacteriological profile in patients with pemphigus.<\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>Materials and Methods:<\/jats:title>\n                    <jats:p>Pus for culture and sensitivity was collected from clinically infected lesions of pemphigus patients in the Department of Dermatology, St. John's Medical College Hospital, Bengaluru, from June 2013 to June 2014.<\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>Results:<\/jats:title>\n                    <jats:p>\n                      Of the 49 patients included in the study, 44 were suffering from pemphigus vulgaris, 4 from pemphigus foliaceus and one had pemphigus vegetans. There were 31 male and 18 female patients. The mean age of the group was 35.51 year. Mean Autoimmune Bullous Disorder Intensity Score was 17.36. About 32.7% were diabetic. About 40.81% showed the growth of\n                      <jats:italic toggle=\"yes\">Staphylococcus aureus<\/jats:italic>\n                      , 12.24% of\n                      <jats:italic toggle=\"yes\">Pseudomonas aeruginosa<\/jats:italic>\n                      , 6.12% of\n                      <jats:italic toggle=\"yes\">Proteus mirabilis<\/jats:italic>\n                      , 4.08% of \u03b2-hemolytic streptococci and nonfermenting Gram-negative bacilli, and 2.04% of\n                      <jats:italic toggle=\"yes\">Proteus vulgaris, Staphylococcus saprophyticus, Enterococcus<\/jats:italic>\n                      species, and\n                      <jats:italic toggle=\"yes\">Klebsiella<\/jats:italic>\n                      species.\n                      <jats:italic toggle=\"yes\">S. aureus<\/jats:italic>\n                      showed 100% sensitivity to antibiotics \u2013 tetracycline, amikacin, chloramphenicol, and netilmicin; 90% resistance was found for penicillin and 55% resistance was found for ciprofloxacin and erythromycin. Methicillin-resistant\n                      <jats:italic toggle=\"yes\">S. aureus<\/jats:italic>\n                      was 30%.\n                      <jats:italic toggle=\"yes\">P. aeruginosa<\/jats:italic>\n                      showed 100% sensitivity to ciprofloxacin, amikacin, gentamicin, piperacillin, piperacillin + tazobactam, and netilmicin.\n                    <\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>Conclusion:<\/jats:title>\n                    <jats:p>\n                      <jats:italic toggle=\"yes\">S. aureus<\/jats:italic>\n                      was the most common organism showing sensitivity to tetracycline, amikacin, chloramphenicol, and netilmicin and resistance to penicillin, ciprofloxacin, and erythromycin.\n                    <\/jats:p>\n                  <\/jats:sec>","DOI":"10.4103\/ijd.ijd_152_17","type":"journal-article","created":{"date-parts":[[2018,7,9]],"date-time":"2018-07-09T01:34:59Z","timestamp":1531100099000},"page":"301-304","update-policy":"https:\/\/doi.org\/10.1097\/lww.0000000000001000","source":"Crossref","is-referenced-by-count":11,"title":["Cutaneous Bacteriological Profile in Patients with Pemphigus"],"prefix":"10.4103","volume":"63","author":[{"given":"K C","family":"Kiran","sequence":"first","affiliation":[{"name":"From the Department of DVL, Dr. BR Ambedkar Medical College Hospital, Bengaluru, Karnataka, India"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"J","family":"Madhukara","sequence":"additional","affiliation":[{"name":"Department of DVL, St. John's Medical College Hospital, Bengaluru, Karnataka, India"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Anil","family":"Abraham","sequence":"additional","affiliation":[{"name":"Department of DVL, St. John's Medical College Hospital, Bengaluru, Karnataka, India"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"S","family":"Muralidharan","sequence":"additional","affiliation":[{"name":"Department of Microbiology, St. John's Medical College Hospital, Bengaluru, Karnataka, India"}],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"276","reference":[{"key":"R1-20250920","doi-asserted-by":"crossref","first-page":"1219","DOI":"10.1016\/S0190-9622(88)70128-0","article-title":"Pemphigus","volume":"18","author":"Korman","year":"1988","journal-title":"J Am Acad Dermatol"},{"key":"R2-20250920","first-page":"89","article-title":"Bacterial culture and sensitivity in pemphigus","volume":"63","author":"Solanki","year":"1997","journal-title":"Indian J Dermatol Venereol Leprol"},{"key":"R3-20250920","doi-asserted-by":"crossref","first-page":"221","DOI":"10.1016\/S0190-9622(82)70111-2","article-title":"Death in pemphigus","volume":"7","author":"Ahmed","year":"1982","journal-title":"J Am Acad Dermatol"},{"key":"R4-20250920","first-page":"1","article-title":"Evaluation of secondary bacterial infection of skin diseases in- and outpatient and their sensitivity to antimicrobials","volume":"3","author":"Abudullah","year":"2007","journal-title":"Egypt Dermatol Online J"},{"key":"R5-20250920","doi-asserted-by":"crossref","first-page":"141","DOI":"10.4103\/0378-6323.77452","article-title":"Clinical and bacteriological profile and outcome of sepsis in dermatology ward in tertiary care center in New Delhi","volume":"77","author":"Asati","year":"2011","journal-title":"Indian J Dermatol Venereol Leprol"},{"key":"R6-20250920","doi-asserted-by":"crossref","first-page":"774","DOI":"10.4103\/0378-6323.102392","article-title":"Etiology of pyrexia in pemphigus patients: A dermatologist's enigma","volume":"78","author":"Qadim","year":"2012","journal-title":"Indian J Dermatol Venereol Leprol"},{"key":"R7-20250920","first-page":"834295","article-title":"Pemphigus vulgaris and infections: A retrospective study on 155 patients","volume":"2013","author":"Esmaili","year":"2013","journal-title":"Autoimmune Dis"},{"key":"R8-20250920","first-page":"413","article-title":"Severity score indexes for blistering diseases","volume":"42","author":"Daniel","year":"2003","journal-title":"Clin Dermatol"},{"key":"R9-20250920","first-page":"5","article-title":"Pemphigus in India. A note","volume":"18","author":"Sehgal","year":"1972","journal-title":"Indian J Dermatol"},{"key":"R10-20250920","first-page":"126","article-title":"A clinicopathological study of pemphigus","volume":"39","author":"Singh","year":"1973","journal-title":"Indian J Dermatol Venereol"},{"issue":"Suppl 1","key":"R11-20250920","doi-asserted-by":"crossref","first-page":"108","DOI":"10.1159\/000246946","article-title":"Pemphigus in oxford, UK, and New Delhi, India: A comparative study of disease characteristics and HLA antigens","volume":"189","author":"Wilson","year":"1994","journal-title":"Dermatology"},{"key":"R12-20250920","doi-asserted-by":"crossref","first-page":"A379","DOI":"10.1016\/j.autrev.2009.10.009","article-title":"Geoepidemiologic considerations of auto-immune pemphigus","volume":"9","author":"Meyer","year":"2010","journal-title":"Autoimmun Rev"},{"key":"R13-20250920","doi-asserted-by":"crossref","first-page":"439","DOI":"10.4103\/0378-6323.82396","article-title":"Pemphigus in India","volume":"77","author":"Kanwar","year":"2011","journal-title":"Indian J Dermatol Venereol Leprol"},{"key":"R14-20250920","doi-asserted-by":"crossref","first-page":"285","DOI":"10.4103\/0378-6323.136828","article-title":"Treatment of pemphigus: An Indian perspective","volume":"80","author":"Kanwar","year":"2014","journal-title":"Indian J Dermatol Venereol Leprol"},{"key":"R15-20250920","first-page":"75","article-title":"A clinicopathological study of pemphigus","volume":"46","author":"Huda","year":"2001","journal-title":"Indian J Dermatol"}],"container-title":["Indian Journal of Dermatology"],"original-title":[],"language":"en","link":[{"URL":"https:\/\/www.ovid.com\/00076423-201863040-00005","content-type":"unspecified","content-version":"vor","intended-application":"similarity-checking"}],"deposited":{"date-parts":[[2026,8,27]],"date-time":"2026-08-27T13:48:48Z","timestamp":1787838528000},"score":1,"resource":{"primary":{"URL":"https:\/\/www.ovid.com\/00076423-201863040-00005"}},"subtitle":[],"short-title":[],"issued":{"date-parts":[[2018,7]]},"references-count":15,"journal-issue":{"issue":"4","published-print":{"date-parts":[[2018]]}},"URL":"https:\/\/doi.org\/10.4103\/ijd.ijd_152_17","relation":{},"ISSN":["0019-5154","1998-3611"],"issn-type":[{"value":"0019-5154","type":"print"},{"value":"1998-3611","type":"electronic"}],"subject":[],"published":{"date-parts":[[2018,7]]},"assertion":[{"value":"2017-03-01","name":"received","label":"Received","group":{"name":"publication_history","label":"Publication History"}},{"value":"2018-02-01","name":"accepted","label":"Accepted","group":{"name":"publication_history","label":"Publication History"}}]}}