{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,8,26]],"date-time":"2026-08-26T19:26:52Z","timestamp":1787772412516,"version":"build-2784847793"},"reference-count":10,"publisher":"Ovid Technologies (Wolters Kluwer Health)","issue":"6","content-domain":{"domain":["lww.com","ovid.com"],"crossmark-restriction":true},"short-container-title":[],"published-print":{"date-parts":[[2022,11]]},"abstract":"<jats:sec>\n                    <jats:title\/>\n                    <jats:p>Alopecia areata (AA) is type of autoimmune, T-cell-mediated disease with abnormal expression of MHC Class I, a common reason for non-scarring hair loss. Familial Mediterranean fever (FMF) is a hereditary autoinflammatory disease characterized by periodic fever and serositis. Various diseases and conditions that may be related to FMF have been reported. It has been known that patients with FMF are vulnerable to MHC Class I-related diseases. The co-existence of the two MHC Class I group-associated entities, FMF and AA, has not been reported in the literature. Here, we present three cases with AA and FMF and discuss the possible common pathway in their pathogenesis.<\/jats:p>\n                  <\/jats:sec>","DOI":"10.4103\/ijd.ijd_312_22","type":"journal-article","created":{"date-parts":[[2024,5,29]],"date-time":"2024-05-29T12:15:14Z","timestamp":1716984914000},"page":"835","update-policy":"https:\/\/doi.org\/10.1097\/lww.0000000000001000","source":"Crossref","is-referenced-by-count":1,"title":["Is There a Pathogenetic Relationship Between Alopecia Areata and Familial Mediterranean Fever?"],"prefix":"10.4103","volume":"67","author":[{"given":"G\u00fcldehan","family":"At\u0131\u015f","sequence":"first","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Simge S\u00fcel","family":"Ero\u011flu","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Gaye","family":"G\u00fcldiken","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"276","reference":[{"key":"R1-71-20240829","doi-asserted-by":"crossref","first-page":"215","DOI":"10.1007\/s10238-020-00673-w","article-title":"Alopecia areata: A review on diagnosis, immunological etiopathogenesis and treatment options","volume":"21","author":"Sterkens","year":"2021","journal-title":"Clin Exp Med"},{"key":"R2-71-20240829","doi-asserted-by":"crossref","first-page":"726","DOI":"10.1016\/j.autrev.2016.03.008","article-title":"Alopecia areata: Animal models illuminate autoimmune pathogenesis and novel immunotherapeutic strategies","volume":"15","author":"Gilhar","year":"2016","journal-title":"Autoimmun Rev"},{"key":"R3-71-20240829","doi-asserted-by":"crossref","first-page":"1","DOI":"10.1016\/j.jaad.2017.04.1141","article-title":"Alopecia areata: Disease characteristics, clinical evaluation, and new perspectives on pathogenesis","volume":"78","author":"Strazzulla","year":"2018","journal-title":"J Am Acad Dermatol"},{"key":"R4-71-20240829","doi-asserted-by":"crossref","first-page":"253","DOI":"10.3389\/fimmu.2017.00253","article-title":"Familial mediterranean fever: Recent developments in pathogenesis and new recommendations for management","volume":"8","author":"\u00d6zen","year":"2017","journal-title":"Front Immunol"},{"key":"R5-71-20240829","doi-asserted-by":"crossref","first-page":"1591","DOI":"10.3906\/sag-2008-11","article-title":"Familial mediterranean fever, from pathogenesis to treatment: A contemporary review","volume":"50","author":"Tufan","year":"2020","journal-title":"Turkish J Med Sci"},{"key":"R6-71-20240829","doi-asserted-by":"crossref","first-page":"1515","DOI":"10.1056\/NEJMra1103442","article-title":"Review: Alopecia areata","volume":"366","author":"Gilhar","year":"2012","journal-title":"N Engl J Med"},{"key":"R7-71-20240829","doi-asserted-by":"crossref","first-page":"623","DOI":"10.1016\/S0002-9440(10)63151-3","article-title":"Collapse and restoration of MHC Class-I-dependent immune privilege exploiting the human hair follicle as a model","volume":"164","author":"Ito","year":"2004","journal-title":"Am J Pathol"},{"key":"R8-71-20240829","doi-asserted-by":"crossref","first-page":"11","DOI":"10.1186\/s43162-021-00040-5","article-title":"Bridging autoinflammatory and autoimmune diseases","volume":"33","author":"El-Shebiny","year":"2021","journal-title":"Egypt J Intern Med"},{"key":"R9-71-20240829","doi-asserted-by":"crossref","first-page":"2733","DOI":"10.3389\/fimmu.2019.02733","article-title":"FMF Is associated with a wide spectrum of MHC Class I- and allied SpA disorders but not with classical MHC Class II-associated autoimmune disease: Insights from a large cohort study","volume":"10","author":"Watad","year":"2019","journal-title":"Front Immunol"},{"key":"R10-71-20240829","doi-asserted-by":"crossref","first-page":"245","DOI":"10.1159\/000485749","article-title":"Alopecia and associated toxic agents: A systematic review","volume":"4","author":"Yu","year":"2018","journal-title":"Skin Appendage Disord"}],"container-title":["Indian Journal of Dermatology"],"original-title":[],"language":"en","link":[{"URL":"https:\/\/www.ovid.com\/10.4103\/ijd.ijd_312_22","content-type":"unspecified","content-version":"vor","intended-application":"similarity-checking"}],"deposited":{"date-parts":[[2026,8,26]],"date-time":"2026-08-26T18:45:16Z","timestamp":1787769916000},"score":1,"resource":{"primary":{"URL":"https:\/\/www.ovid.com\/10.4103\/ijd.ijd_312_22"}},"subtitle":[],"short-title":[],"issued":{"date-parts":[[2022,11]]},"references-count":10,"journal-issue":{"issue":"6","published-print":{"date-parts":[[2022]]}},"URL":"https:\/\/doi.org\/10.4103\/ijd.ijd_312_22","relation":{},"ISSN":["0019-5154"],"issn-type":[{"value":"0019-5154","type":"print"}],"subject":[],"published":{"date-parts":[[2022,11]]}}}