{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,8,19]],"date-time":"2026-08-19T20:00:08Z","timestamp":1787169608859,"version":"build-2736575974"},"reference-count":9,"publisher":"Ovid Technologies (Wolters Kluwer Health)","issue":"2","content-domain":{"domain":["lww.com","ovid.com"],"crossmark-restriction":true},"short-container-title":[],"published-print":{"date-parts":[[2022,4]]},"abstract":"<jats:sec>\n                    <jats:title\/>\n                    <jats:p>Amelogenesis imperfecta (AI) comprises a complicated group of conditions, involving a structural defect of the tooth enamel, owing to certain genetic disturbances. It is expressed as an autosomal dominant, autosomal recessive or X-linked recessive trait. The structural pattern and clinical appearances of all the teeth, including those in the deciduous as well as the permanent dentition, are affected. It may be differentiated into three main divisions such as hypoplastic, hypocalcific and hypomaturative patterns. Eruption of tooth refers to a complex coordinated physiological process characterized by the movement of a tooth from its initial developmental position within the jaws to its final functional position in the occlusal plane, dictated crucially by dental follicle, bony remodeling metabolic alteration and molecular determinants. Alteration involving any one of the factors might cause hindrances. Herein, we report two cases with AI associated with noneruption.<\/jats:p>\n                  <\/jats:sec>","DOI":"10.4103\/jomfp.jomfp_471_20","type":"journal-article","created":{"date-parts":[[2023,4,26]],"date-time":"2023-04-26T05:27:46Z","timestamp":1682486866000},"page":"254-258","update-policy":"https:\/\/doi.org\/10.1097\/lww.0000000000001000","source":"Crossref","is-referenced-by-count":3,"title":["Noneruption of teeth in amelogenesis imperfecta"],"prefix":"10.4103","volume":"26","author":[{"given":"Jaygopal G","family":"Ray","sequence":"first","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Sayani","family":"Dutta","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Snehanjan","family":"Sarangi","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Priyanka","family":"Yadav","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"276","reference":[{"key":"R3-19-20240906","doi-asserted-by":"crossref","first-page":"377","DOI":"10.1038\/sj.bdj.2012.314","article-title":"Amelogenesis imperfecta: An introduction","volume":"212","author":"Gadhia","year":"2012","journal-title":"Br Dent J"},{"key":"R4-19-20240906","doi-asserted-by":"crossref","first-page":"738","DOI":"10.1177\/0810738","article-title":"Autosomal-dominant hypoplastic form of amelogenesis imperfecta caused by an enamelin gene mutation at the exon-intron boundary","volume":"81","author":"Kida","year":"2002","journal-title":"J Dent Res"},{"key":"R6-19-20240906","doi-asserted-by":"crossref","first-page":"224","DOI":"10.1159\/000151378","article-title":"Human and mouse enamel phenotypes resulting from mutation or altered expression of AMEL, ENAM, MMP20 and KLK4","volume":"189","author":"Wright","year":"2009","journal-title":"Cells Tissues Organs"},{"key":"R8-19-20240906","doi-asserted-by":"crossref","first-page":"19","DOI":"10.1034\/j.1601-0825.2003.00843.x","article-title":"Amelogenesis imperfecta: A classification and catalogue for the 21st century","volume":"9","author":"Aldred","year":"2003","journal-title":"Oral Dis"},{"key":"R9-19-20240906","doi-asserted-by":"crossref","first-page":"e207","DOI":"10.4317\/jced.2.e207","article-title":"Hypoplastic amelogenesis imperfecta with multiple impacted teeth-reports of two cases","volume":"2","author":"Reddy","year":"2010","journal-title":"J Clin Exp Dent"},{"key":"R10-19-20240906","doi-asserted-by":"crossref","first-page":"5","DOI":"10.1186\/s13005-018-0163-7","article-title":"Primary failure of eruption (PFE): A systematic review","volume":"14","author":"Hanisch","year":"2018","journal-title":"Head Face Med"},{"key":"R11-19-20240906","first-page":"26","article-title":"Dental development in amelogenesis imperfecta: A controlled study","volume":"17","author":"Seow","year":"1995","journal-title":"Pediatr Dent"},{"key":"R12-19-20240906","doi-asserted-by":"crossref","first-page":"170","DOI":"10.1590\/S0103-64402010000200014","article-title":"Amelogenesis imperfecta, hypoplastic type associated with some dental abnormalities: A case report","volume":"21","author":"Canger","year":"2010","journal-title":"Braz Dent J"},{"key":"R13-19-20240906","doi-asserted-by":"crossref","first-page":"1","DOI":"10.4103\/ijohr.ijohr_11_18","article-title":"Primary failure of eruption, a review","volume":"4","author":"Dixit","year":"2018","journal-title":"Indian J Oral Health Res"}],"container-title":["Journal of Oral and Maxillofacial Pathology"],"original-title":[],"language":"en","link":[{"URL":"https:\/\/www.ovid.com\/10.4103\/jomfp.JOMFP_471_20","content-type":"unspecified","content-version":"vor","intended-application":"similarity-checking"}],"deposited":{"date-parts":[[2026,8,19]],"date-time":"2026-08-19T19:09:52Z","timestamp":1787166592000},"score":1,"resource":{"primary":{"URL":"https:\/\/www.ovid.com\/10.4103\/jomfp.JOMFP_471_20"}},"subtitle":["A report of two cases and review"],"short-title":[],"issued":{"date-parts":[[2022,4]]},"references-count":9,"journal-issue":{"issue":"2","published-print":{"date-parts":[[2022]]}},"URL":"https:\/\/doi.org\/10.4103\/jomfp.jomfp_471_20","relation":{},"ISSN":["0973-029X"],"issn-type":[{"value":"0973-029X","type":"print"}],"subject":[],"published":{"date-parts":[[2022,4]]}}}