{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,9,22]],"date-time":"2026-09-22T18:48:43Z","timestamp":1790102923102,"version":"4.0.1"},"reference-count":16,"publisher":"Ovid Technologies (Wolters Kluwer Health)","issue":"2","content-domain":{"domain":["lww.com","ovid.com"],"crossmark-restriction":true},"short-container-title":[],"published-print":{"date-parts":[[2023,5]]},"abstract":"<jats:p>\n                    A 32-year-old male with a past history of bilateral laser-assisted\n                    <jats:italic toggle=\"yes\">in situ<\/jats:italic>\n                    keratomileusis (LASIK), presented with a decrease in vision in both eyes (BE). Clinical examination showed diffuse corneal haze and interface fluid syndrome (IFS), which was confirmed on anterior segment optical coherence tomography. Intraocular pressure (IOP) measurements obtained with Goldmann applanation tonometer (GAT) were low; however, digital tonometry indicated a raised IOP in BE, which was confirmed by measuring IOP with GAT, outside the peripheral edge of the LASIK flap. Treatment with antiglaucoma medications resulted in complete resolution of the interface fluid. However, due to noncompliance with medications, it recurred and glaucoma showed progression. The patient underwent trabeculectomy with mitomycin C in BE, resulting in improvement in the visual acuity, clear cornea, resolution of interface fluid, and normal IOP, till the last follow-up visit. This case had IFS secondary to episodes of raised IOP, which was diagnosed on careful clinical examination. IOP readings using GAT over the central cornea can give spuriously low readings in IFS. Hence, IOP should be measured in the peripheral cornea to obtain correct IOP readings. Early detection of high IOP and its management is essential to halt glaucoma progression.\n                  <\/jats:p>","DOI":"10.4103\/ojo.ojo_112_22","type":"journal-article","created":{"date-parts":[[2024,5,24]],"date-time":"2024-05-24T13:56:24Z","timestamp":1716558984000},"page":"329-332","update-policy":"https:\/\/doi.org\/10.1097\/lww.0000000000001000","source":"Crossref","is-referenced-by-count":2,"title":["Bilateral interface fluid syndrome and glaucoma progression after laser-assisted in situ keratomileusis"],"prefix":"10.4103","volume":"16","author":[{"given":"Tarannum","family":"Mansoori","sequence":"first","affiliation":[{"name":"Department of Glaucoma, Anand Eye Institute, Hyderabad, Telangana, India"}],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"276","published-online":{"date-parts":[[2023,3,27]]},"reference":[{"key":"R1-20240908","doi-asserted-by":"crossref","first-page":"1009","DOI":"10.1016\/S0886-3350(99)00083-8","article-title":"Interface fluid 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LASIK the effect of the refractive correction and the lamellar flap","volume":"112","author":"Chang","year":"2005","journal-title":"Ophthalmology"},{"key":"R15-20240908","doi-asserted-by":"crossref","first-page":"1848","DOI":"10.1016\/j.ophtha.2007.01.029","article-title":"Interface fluid syndrome in human eye bank corneas after LASIK:Causes and pathogenesis","volume":"114","author":"Dawson","year":"2007","journal-title":"Ophthalmology"},{"key":"R16-20240908","doi-asserted-by":"crossref","first-page":"1320","DOI":"10.1016\/S0886-3350(98)80222-8","article-title":"Correlation of intraocular pressure and central corneal thickness in normal myopic eyes and after laser in situ keratomileusis","volume":"24","author":"Emara","year":"1998","journal-title":"J Cataract Refract Surg"}],"container-title":["Oman Journal of 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