{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,7,8]],"date-time":"2026-07-08T11:15:05Z","timestamp":1783509305497,"version":"3.55.0"},"reference-count":89,"publisher":"MDPI AG","issue":"5","content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":[],"published-print":{"date-parts":[[2025,9]]},"abstract":"<jats:p>\n                    <jats:bold>Background:<\/jats:bold>\n                    In patients with rheumatoid arthritis (RA), the pathologic progression of lower-limb biomechanics is established. Although specific aspects of RA gait patterns have been studied and described, we are aware of no studies of gait pattern compensations across the entire disease course. This study aimed to describe a model that could predict the evolution of lower-limb pathomechanics in patients with RA.\n                  <\/jats:p>\n                  <jats:p>\n                    <jats:bold>Methods:<\/jats:bold>\n                    A literature review was conducted of electronic databases (MEDLINE, PEDro, Trip Database, DOAJ, BioMed Central, PLoS Clinical Trials, ScienceDirect, CRD York University, AHRQ, NICE, and Cochrane Library) to October 3, 2023.\n                  <\/jats:p>\n                  <jats:p>\n                    <jats:bold>Results:<\/jats:bold>\n                    A theory was developed that all people with RA induce or augment gait evolution syndromes following the same biomechanical course. Specifically, we postulate rheumatoid equinus syndrome, rheumatoid abnormal pronation syndrome, and rheumatoid shuffle syndrome, which have never been described before.\n                  <\/jats:p>\n                  <jats:p>\n                    <jats:bold>Conclusions:<\/jats:bold>\n                    A new model of the evolution of gait compensation in RA is proposed. An important challenge of RA is that it increases the risk of ulcerative lesions, falls, pain, fractures, and health-care costs. The proposed model can be used to reduce morbidity in this patient group by helping explain and reduce the pain, deformity, and ankylosis of foot RA.\n                  <\/jats:p>","DOI":"10.7547\/23-020","type":"journal-article","created":{"date-parts":[[2024,8,15]],"date-time":"2024-08-15T11:16:39Z","timestamp":1723720599000},"source":"Crossref","is-referenced-by-count":0,"title":["Theory of the Biomechanical Evolution of the Rheumatoid Foot: A Narrative Review"],"prefix":"10.3390","volume":"115","author":[{"given":"Gaetano Di","family":"Stasio","sequence":"first","affiliation":[{"name":"Podos Logo Italia, Naples, Italy."}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Carles","family":"Verg\u00e9s-Salas","sequence":"additional","affiliation":[{"name":"Department of Clinical Sciences, School of Podiatry, Faculty of Medicine and Health Sciences, University of Barcelona, Barcelona, Spain."}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Elena de","family":"Planell-Mas","sequence":"additional","affiliation":[{"name":"Department of Clinical Sciences, School of Podiatry, Faculty of Medicine and Health Sciences, University of Barcelona, Barcelona, Spain."}],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"1968","reference":[{"key":"B1","series-title":"Nat Rev Rheumatol","first-page":"9","article-title":"Anti-inflammatory and immune-regulatory cytokines in rheumatoid arthritis","volume":"15","author":"Chen Z","year":"2019","unstructured":"Chen Z, Bozec A, Ramming A, et al: Anti-inflammatory and immune-regulatory cytokines in rheumatoid arthritis. 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