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Kusters JG, van Vliet AHM, Kuipers EJ. Pathogenesis of <i>Helicobacter pylori<\/i> Infection. Clin Microbiol Rev. 2006;19:449-490.","DOI":"10.1128\/CMR.00054-05"},{"key":"2","doi-asserted-by":"crossref","unstructured":"2. El Khadir M, Alaoui Boukhris S, Benajah D-A, et al. <i>VacA<\/i> and <i>CagA<\/i> status as biomarker of two opposite end outcomes of <i>Helicobacter pylori<\/i> infection (gastric cancer and duodenal ulcer) in a Moroccan population. PLoS One. 2017;12:e0170616.","DOI":"10.1371\/journal.pone.0170616"},{"key":"3","doi-asserted-by":"crossref","unstructured":"3. Alaoui Boukhris S, Amarti A, El Rhazi K, et al. <i>Helicobacter pylori<\/i> genotypes associated with gastric histo-pathological damages in a Moroccan population. PLoS One. 2013;8:e82646.","DOI":"10.1371\/journal.pone.0082646"},{"key":"4","doi-asserted-by":"crossref","unstructured":"4. El Khadir M, Alaoui Boukhris S, Benajah D-A, et al. <i>Helicobacter pylori<\/i> CagA EPIYA-C motifs and gastric diseases in Moroccan patients. Infect Genet Evol. 2018;66.120-129.","DOI":"10.1016\/j.meegid.2018.09.015"},{"key":"5","doi-asserted-by":"crossref","unstructured":"5. El Khadir M, Alaoui Boukhris S, Benajah D-A, et al. <i>VacA<\/i> genotypes and cagA\u2010EPIYA\u2010C motifs of <i>Helicobacter pylori<\/i> and gastric histopathological lesions. Int J Cancer. 2020;147:3206-3214.","DOI":"10.1002\/ijc.33158"},{"key":"6","doi-asserted-by":"crossref","unstructured":"6. Ogiwara H, Sugimoto M, Ohno T, et al. Role of deletion located between the intermediate and middle regions of the <i>Helicobacter pylori vacA<\/i> gene in cases of gastroduodenal diseases. J Clin Microbiol. 2009;47:3493-3500.","DOI":"10.1128\/JCM.00887-09"},{"key":"7","doi-asserted-by":"crossref","unstructured":"7. Alam J, Sarkar A, Karmakar BC, et al. Novel virulence factor <i>dupA<\/i> of <i>Helicobacter pylori<\/i> as an important risk determinant for disease manifestation: An overview. World J Gastroenterol. 2020;26:4739-4752.","DOI":"10.3748\/wjg.v26.i32.4739"},{"key":"8","doi-asserted-by":"crossref","unstructured":"8. Phuc BH, Tuan VP, Dung HDQ, et al. <i>Helicobacter pylori<\/i> type 4 secretion systems as gastroduodenal disease markers. Sci Rep. 2021;11:4584.","DOI":"10.1038\/s41598-021-83862-1"},{"key":"9","doi-asserted-by":"crossref","unstructured":"9. Lu H, Hsu P-I, Graham DY, et al. Duodenal ulcer promoting gene of <i>Helicobacter pylori<\/i>. Gastroenterology. 2005;128:833-848.","DOI":"10.1053\/j.gastro.2005.01.009"},{"key":"10","doi-asserted-by":"crossref","unstructured":"10. Alaoui Boukhris S, Benajah D -a., El Rhazi K, et al. Prevalence and distribution of <i>Helicobacter pylori cagA<\/i> and <i>vacA<\/i> genotypes in the Moroccan population with gastric disease. Eur J Clin Microbiol Infect Dis. 2012; 31:1775-1781. 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Mitchell DJ, Huynh HQ, Ceponis PJM, et al. <i>Helicobacter pylori<\/i> disrupts STAT1-mediated gamma interferon-induced signal transduction in epithelial cells. Infect Immun. 2004;72:537-545.","DOI":"10.1128\/IAI.72.1.537-545.2004"},{"key":"30","doi-asserted-by":"crossref","unstructured":"30. Talebi Bezmin Abadi A, Taghvaei T, Wolfram L, et al. Infection with <i>Helicobacter pylori<\/i> strains lacking <i>dupA<\/i> is associated with an increased risk of gastric ulcer and gastric cancer development. J Med Microbiol. 2012;61:23-30.","DOI":"10.1099\/jmm.0.027052-0"},{"key":"31","doi-asserted-by":"crossref","unstructured":"31. Dean AE, Reichardt F, Anakk S. Sex differences feed into nuclear receptor signaling along the digestive tract. 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